The RheumNow Week in Review – 12 May 2017-YouTube Save
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I can't help but wonder if the reported increase in heart disease associated with active RA is really due to increased inflammation affecting plaque formation or the effects of NSAIDs that have been widely used in the past as background therapy for DMARDs.
Another condition for whuch we have little therapy and is possibly related to inflammatory OA is CPPD disease. All we really have is steroids for pseudogout even though colchicine and DMARDs are used in desperation, not to mention Plaquenil.
I appreciate your concise reports.
David, thanks for the thoughts. OA needs a big push from all of us. RA and CVD I believe is largely driven by both inflammation and preclinical pathology that may be related to ACPA and other factors we link with bad RA, those just happen to be proatherogenic too! - Jack
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